Well both dogs and cats get mucosal striations and when they coalesce I dubbed this phenomenon a few years ago “mucosal fogging” because the striations get obscured as they group together. In dogs this is almost pathogneumonic for lymphangectasia and PLE but in cats its still up in the air but I see it with chronic maldigestion losing weight for no reason cats with or without GI signs.
Well both dogs and cats get mucosal striations and when they coalesce I dubbed this phenomenon a few years ago “mucosal fogging” because the striations get obscured as they group together. In dogs this is almost pathogneumonic for lymphangectasia and PLE but in cats its still up in the air but I see it with chronic maldigestion losing weight for no reason cats with or without GI signs. The cat title image is mine but my friend Doug Casey DABVP of Vancouver sent me this nerd candy image of mucosal fogging in a PLE dog and thought it would be a cool one to put up on a forum post to start the weekend. I surely going to look at fried foods differently this weekend now lol.
Anyone have any input on the mucosal striations/fogging in cats like the title image above? I haven’t seen any new research out on it bu that’s where I defer to the literature Guru of the team remo Lobetti. I’ll ping him. Happy Friday and weekend everyone!
Comments
Nothing reported/described in
Nothing reported/described in the cat but as mucosal striations (dogs) are associated with mucosal inflammation and protein losing enteropathy, no reason why similar lesions cannot be present in the cats with IBD, pancreatic insufficiency, and lymphoma.
I had a case of mucosal
I had a case of mucosal ‘fogging’ and generalised thickening of small intestinal wall in an 11 year old lab x boxer male neutered dog this week.
Presenting signs of diarrhoea and hypoalbuminaemia.
Why would lymphangiectasia occur in an older dog, would you be worried about underlying lymphoma?
In my experience i can only
In my experience i can only make generalizations on PLE with LSA which happens… typically non PLE breed patients and not the usual PLE breed like yorkies. Usually in PLE + LSA cases loss of mural detail occurs and regional LN are involved whereas standard yorkie type PLE the submucosal layer is in tact and the problem is all mucosal… i.e. mucosal fogging and striations. This is a major generalizaiton but its a point of reference to start from. When there are LN involved especially if distorted and no longer hold solid length to width ratios then think LSA and really push for sampling.