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One-year 6-month old scottish fold male was presented for lameness examination of the hind limb. Grade 2 parasternal heart murmur was heard. The cats seems to have HCM phenotype, probably just the beginning. There seems to be systolic anterior motion also. Can you please confirm if there is SAM and this cat has HCM phenotype? The LVOT velocity was 2.2m/sec. False tendon is clearly visible in the 4-chambered long axis and sort axis view of the apex
Comments
Looks like there is a false
Looks like there is a false tendon in this LV and i got sam on a video you had in the telemed image set you sent (still attached). Its minor but there. Given the lamness be sure to check the iliac trifurcation with CF to ensure no thrombus starting as they dont have to be in failure to throw or form a thrombus. SAM can come and go and best seen with elevated HR.
Thanks for the reply Erric.
Thanks for the reply Erric. We are planning to incprporate more telemed services of sonopath in our practice, so I thought it will be good to start with this
How about HCM? starting HCM?
Can SAM exist without mitral regurgitation?
I didn’t see an MR jet on CF doppler
Yes officially “HCM
Yes officially “HCM phenotype” is the current correct terminology because the ivsd and pwd are > 6 mm. The MR is likely there just need the correct angle of incidence to get it which can be tricky if the jet is small and close down the CF sector to just encompass the MV 1/3 to the left of the valve and 2/3 to the right of the valve and try sdep 1 position and sdep 4 (apical) to find it. The murmur comes from mr and lvot turbulence typically.
Cam anything be done to
Cam anything be done to prevemt HCM from developing further? Atenolol?
atenolol would be the tx but
atenolol would be the tx but as to the effectiveness? or preventive potential? That’s up in the air… throw that quesiton out to a group of cardiologists at acvim and watch the games begin