Kelly and Suzanne,

I think this is the document you were looking for. 

I hope this helps. I did not know how to attach a .pdf so I am posting it here and I will e mail Kelly the .pdf document for anyone that is intereste. I hope this helps for a reference. Maybe Peter can correct any mistakes.

L Atrium:

Too large:

1. L ventricular systolic dysfunction (e.g. DCM)

2. Left ventricular diastolic dysfunction (e.g. HCM)

Kelly and Suzanne,

I think this is the document you were looking for. 

I hope this helps. I did not know how to attach a .pdf so I am posting it here and I will e mail Kelly the .pdf document for anyone that is intereste. I hope this helps for a reference. Maybe Peter can correct any mistakes.

L Atrium:

Too large:

1. L ventricular systolic dysfunction (e.g. DCM)

2. Left ventricular diastolic dysfunction (e.g. HCM)

3. Mitral valvular disease (e.g. endocardiosis)

4. L to R shunting

5. Iatrogenic (e.g. fluid overload)

To small:

1. Hypovolemia

Not empty:

1. Thrombus (cats>dogs)

2. Spontaneous echo contrast (e.g. “smoke”)

3. Mass (e.g. neoplasia, abscess)

 

L Ventricle:

Too large:

1. Volume overload (e.g. DCM, endocardiosis, L to R shunt)

2. High output state (e.g. anemia, hyperthyroidisim)

3. Iatrogenic (fluid overload)

Too small:

1. Hypovolemia

2. Severe concentric LV hypertrophy (e.g. HCM)

3. Right ventricular volume or pressure overload

 

LV Posterior Wall:

Too thick:

1. Outflow obstruction (e.g. systemic hypertension, SAS)

2. Inappropriate hypertrophy (e.g. HCM, RCM)

3. Infiltrative disease

Too thin, poor motion:

1. DCM

2. Prior myocardial infarction

Too Bright:

1. ischemia and/or fibrosis

 

Interventricular Septum:

Too thick:

1. Outflow obstruction (e.g. systemic hypertension, SAS)

2. Inappropriate hypertrophy (e.g. HCM, RCM)

3. Infiltrative disease

4. Right ventricular outflow obstruction (e.g. PS, PH)

Too thin, poor motion:

1. DCM

2. Prior myocardial infarction

Too bright:

1. ischemia and/or fibrosis

 

Piece Missing:

1. VSD

 

Flattening:

Right ventricular pressure or volume overload

 

Paradoxical Septal Motion:

1. RV overload as for flattening

2. Conduction disturbances

 

Global LV Function:

Increased:

1. High output states (e.g. anemia, fever, increased T4)

2. Hypertrophic Cardiomyopathy

3. Mitral insufficiency (“hyper-dynamic”)

Decreased:

1. DCM

2. Restrictive cardiomyopathy (some)

3. Severe hypothyroidism

4. Sepsis

5. Concurrent tachyarrythmia

 

LFOT/Aortic Valve:

LV outflow tract too narrow (visible ridge)

1. SAS

Thickened valve +/- abnormal valve movement

1. Valvular aortic stenosis

2. Aortic endocarditis

 

Aorta:

Too wide:

1. Post stenotic dilation

2. Systemic hypertension

Too narrow:

1. Hypovolemia

2. Poor Cardiac output (e.g. DCM)

 

Right Atrium:

Too large:

1. Tricuspid valvular disease (e.g. endocardiosis, TV dysplasia

2. Atrial septal defect

3. Iatrogenic (fluid overload)

Too small:

1. Hypovolemia

2. Collapse due to pericardial effusion/cardiac tamponade

 

Tricuspid Valve:

Too Thick:

1. Tricuspid valvular dysplasia (+/- long, tethered appearance)

2. Chronic degenerative valvular disease (endocardiosis)

Abnormal motion:

1. Tricuspid stenosis- restricted movement during diastole

 

Multiple double lines crossing valve:

Heartworm disease

 

Right Ventricle:

Too large:

1. Volume overload (e.g. DCM, tricuspid insufficiency)

2. Iatrogenic (fluid overload)

Too small:

1. Hypovolemia

2. Cardiac tamponade

 

Thickened Walls:

1. Pressure overload (e.g. PS, Pulmonary hypertension

2. Infiltrative disease

 

Pulmonic Valve and PA:

Thickened Valve:

1. Pulmonic stenosis

Dilated Pulmonary artery:

1. Post-stenotic dilation

2. Pulmonary hypertension