- 13 year old Maltese Terrier presented with vomiting and acting strange, previous hx GME (not on any meds now)
- Bloods: severely increased ALP + ALT (proportionate >3000), GLDH normal, resting bile acids off scale (but is a Maltese), bilirubin normal, Albumin mild decrease, no stress leukogram
- The liver seemed mildly heterogenous with increased echogenicity, cystic structure and abnormal gallbladder (FNA liver pending)
- The stomach layering seemed abnormal – how would you describe this?
- 13 year old Maltese Terrier presented with vomiting and acting strange, previous hx GME (not on any meds now)
- Bloods: severely increased ALP + ALT (proportionate >3000), GLDH normal, resting bile acids off scale (but is a Maltese), bilirubin normal, Albumin mild decrease, no stress leukogram
- The liver seemed mildly heterogenous with increased echogenicity, cystic structure and abnormal gallbladder (FNA liver pending)
- The stomach layering seemed abnormal – how would you describe this?
- Gastric and pancreaticoduodenal lymph nodes were mildly enlarged
- A right adrenal mass was present (no pu/pd, lean body condition) ddx adenoma…functional? or phaeo?
- What do you think is driving the severe liver enzyme elevations?
- Thanks in advance
Comments
A number of things can drive
A number of things can drive LEs. Consider Lepto if in your area but needs a needle fna for sure if not core bx.