Dear Colleagues, I would much appreciate your help with these videos.
4 m. old MI bengal cat, seen 3 days ago for a 2 months history of self trauma on tip of tail. Now the last 3cm of tail present signs of ischemic necrosis (dark skin, cold, few small scabs). At presentation, BP and SpO2 where not tested.
Cat belongs to a multicat (5 other) household, purely indoor. Cat is FeLV+/FIV-, has no other symptomatology. rest of cats are FeLV -.
Echocardio: no overt signs of cardiac disease
Dear Colleagues, I would much appreciate your help with these videos.
4 m. old MI bengal cat, seen 3 days ago for a 2 months history of self trauma on tip of tail. Now the last 3cm of tail present signs of ischemic necrosis (dark skin, cold, few small scabs). At presentation, BP and SpO2 where not tested.
Cat belongs to a multicat (5 other) household, purely indoor. Cat is FeLV+/FIV-, has no other symptomatology. rest of cats are FeLV -.
Echocardio: no overt signs of cardiac disease
Tail scan: videos show the ventral artery on both sagittal and transverse view, aprox 1.5-2cm from base of tail, where doppler seems to be interrupted partially. In one of the videos there seems to be a curvature in the vessel which now makes me think of an external mass effect compression rather than an intravascular embolus of some sort. From mid-tail distally my probe does not manage to find any doppler activity,however, the SpO2 was positive for pulse and O2 saturation on tip of tail was 100% just yesterday. I would like some reassurance on my findings.
DDx: I am considering: trauma related compression of artery (soft tissue inflammation…others?),thromboembolic disease (probably not heart related)-hypercoagulation state?, other type of embolus (bacterial, could it be potentially related to FeLV infection in some way? inmunecomplexes?). The question is whether the biting came first due to a vascular problem or second to an initial behavioral issue…It could also mean cold agglutination disease but as per today routine CBC has not been done yet. I practically rule out neoplastic disease (vascular or metastatic) due to age.
For now my colleagues have him on Pen G injs, amoxicalv oral, metacam 0.05mg/kg Sid for 10 days. started yesterday after I did scan. (abs were from start). I feel tempted to investigate further on cold aglutination and wait to see if block improves with given therapy before considering any amputation. Does this sound reasonable?
Comments
Im seeing pretty solid blood
Im seeing pretty solid blood flow here..Sounds like he’s heading to amputation though.
Im seeing pretty solid blood
Im seeing pretty solid blood flow here..Sounds like he’s heading to amputation though.
EL…thank you for your
EL…thank you for your input…I thought the interruption in the flow was very obvious when I was scanning…There seemed to be a clear stop in the flow right in the middle of the doppler window…which re-started vaguely after the said area…both on sagittal and on transverse views…How can I differenciate better for next time? It is the second time i think I can see clearly an obstructed artery but specialists tell me there’s not:(( I must be missing something on the technique…
EL…thank you for your
EL…thank you for your input…I thought the interruption in the flow was very obvious when I was scanning…There seemed to be a clear stop in the flow right in the middle of the doppler window…which re-started vaguely after the said area…both on sagittal and on transverse views…How can I differenciate better for next time? It is the second time i think I can see clearly an obstructed artery but specialists tell me there’s not:(( I must be missing something on the technique…
There may just be signal
There may just be signal interruption in that empty spot but ischemia doesnt pick up again after the obstruction/clot…When thrombus is present you can see an echogenicity (not present here) the signal drops completely distally or diminishes solidly. Here the signal is essentially equal on both sides of the drop out. if there are collaterals formed like in a splenic thrombus then this changes a bit but with the coccygeal artery not much opportunity for collateral circulation… its a dead end (no pun intended) so there should be minimal to no distal CF signal if a vascular obstruction were present.
Take a look on the basic search for “thrombus” here.
http://sonopath.com/members/case-studies/search?text=thrombus&species=All
There may just be signal
There may just be signal interruption in that empty spot but ischemia doesnt pick up again after the obstruction/clot…When thrombus is present you can see an echogenicity (not present here) the signal drops completely distally or diminishes solidly. Here the signal is essentially equal on both sides of the drop out. if there are collaterals formed like in a splenic thrombus then this changes a bit but with the coccygeal artery not much opportunity for collateral circulation… its a dead end (no pun intended) so there should be minimal to no distal CF signal if a vascular obstruction were present.
Take a look on the basic search for “thrombus” here.
http://sonopath.com/members/case-studies/search?text=thrombus&species=All
Consider some sort of cauda
Consider some sort of cauda equina syndrome with compression of nerves causing self trauma.
I agree with EL- this cat is probably heading to surgery.
Consider some sort of cauda
Consider some sort of cauda equina syndrome with compression of nerves causing self trauma.
I agree with EL- this cat is probably heading to surgery.
Thank you EL and Randy for
Thank you EL and Randy for your time and input. Very much appreciated.
I hope we can get to the bottom of this case. I hope I have the chance to re-scan the tail again just before surgery (if that is what it ends up being).
I will keep you posted. Thanks again.
Thank you EL and Randy for
Thank you EL and Randy for your time and input. Very much appreciated.
I hope we can get to the bottom of this case. I hope I have the chance to re-scan the tail again just before surgery (if that is what it ends up being).
I will keep you posted. Thanks again.