A 10-year-old NM DSH with a history of diabetes and currently on 6 IU Lantus was presented for revaluation of “not being right”. Abnormalities on CBC and serum biochemistry were neutrophilia, elevated BUN 32, and mild hypoglycemia (68).
A 10-year-old NM DSH with a history of diabetes and currently on 6 IU Lantus was presented for revaluation of “not being right”. Abnormalities on CBC and serum biochemistry were neutrophilia, elevated BUN 32, and mild hypoglycemia (68).
Dystrophic kidneys with calculi.
Comet tail infarct in the right kidney and multiple infarcts in the left kidney.
Pyloric thickening with epigastric lymphadenopathy.
Stomach revealed some progressively shadowing material, potential hairball accumulation. Localized pyloric thickening was noted that measured 0.6 cm with reactive inflamed, surrounding fat and epigastric lymphadenopathy that measured 0.58 cm. The ileocecal region was free of evident pathology.
The left kidney in this patient was severely dystrophic with cortical infarcts and minor pyelectasia that measured 0.7 cm. The left kidney was subnormal in size at 2.2 cm. The right kidney revealed moderate dystrophic changes, cortical infarcts and pelvic calculi. The patient is likely passing calculi in obstructive patterns periodically. The right kidney measured 4.23 cm. Comet tail infarct was noted at the cranial pole of the right kidney with an adjacent corticomedullary calculus noted, which measured 0.88 cm and was non obstructive at this time. However, this is “dragging” infarcting parenchyma along with it, in that portion of the cortex. The infarct is likely owing to stone passage.
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Resolving diabetes mellitus, pancreatitis, pancreatic neoplasia, GI hemorrhage (ulcer, IBD, neoplasia, foreign body)
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