A 14-year-old FS Bengal cat with history of polycystic kidneys was presented for hematuria. Physical exam was unremarkable. Blood chemistry revealed slight hyperamylasemia. CBC was within normal limits. In-house urinalysis showed normal pH, specific gravity and a full field of RBCs on microscopic evaluation. Patient was presented several weeks later for vocal changes, including loss of voice, accompanied by wheezing. Physical exam at that time found the patient to be febrile. Her throat palpated normally. Blood chemistry revealed only high triglycerides.

A 14-year-old FS Bengal cat with history of polycystic kidneys was presented for hematuria. Physical exam was unremarkable. Blood chemistry revealed slight hyperamylasemia. CBC was within normal limits. In-house urinalysis showed normal pH, specific gravity and a full field of RBCs on microscopic evaluation. Patient was presented several weeks later for vocal changes, including loss of voice, accompanied by wheezing. Physical exam at that time found the patient to be febrile. Her throat palpated normally. Blood chemistry revealed only high triglycerides. CBC found leukocytosis and neutrophilia. T4 was within normal range. Urinalysis showed normal pH and specific gravity, a turbid red appearance, proteinuria (3+), hematuria (3+), WBC (4-10), RBC (51-100), and moderate amorphous debris. Urine microalbumin was high. Urine culture yielded no growth. Antibiotic injection and subcutaneous fluids were given, and patient was discharged. Upon recheck exam a few days later, patient was BAR, with slightly tacky mucous membranes, prominent kidneys, mild ocular discharge, heart and lungs clear with no overt murmurs or wheezes ausculted. Thoracic radiographs showed no abnormalities. Patient received subcutaneous fluids, an antibiotic injection, and was referred for ultrasound.