Hi there
- This is a 12 year old FS boxer mix dog with recent onset hematemesis.
- Dog has a PCV of 25%, ALP is>1000, ALT mildly elevated. BUN elevated at 58, creat normal.
- Clotting times pending.
- No other overt abnormalities, some shadowing material in stomach but dog had eaten this morning. I did not see any clots.
- Would these be considered acquired shunts? What does this indicate?
- Thanks for any help,
Liz
Hi there
- This is a 12 year old FS boxer mix dog with recent onset hematemesis.
- Dog has a PCV of 25%, ALP is>1000, ALT mildly elevated. BUN elevated at 58, creat normal.
- Clotting times pending.
- No other overt abnormalities, some shadowing material in stomach but dog had eaten this morning. I did not see any clots.
- Would these be considered acquired shunts? What does this indicate?
- Thanks for any help,
Liz
Comments
From the history looks like
From the history looks like acute hepatopathy with either primary or secondary gastric ulceration/gastric hemorrhage. Not sure about the hepatic vessel and may be an incidental finding – acquired shunts usually associated with chronic liver disease rather than acute disease.
There were multiple vessels
There were multiple vessels like this throughout an enlarged and hyperechoic liver. Does that make a difference?
This is odd because acquired
This is odd because acquired shunts are typically extrahepatic with ascites and edematous pancreas and spleen. Im thinking intrahepatic vascular anomaly that this dog has been living with and now acute inflammatory disease.
CT with contrast would be the way to go here
Would get the liver and
Would get the liver and ulcer/hemorrhage under control before doing anesthesia/sedation for the CT.
CT won’t ever happen. Limited
CT won’t ever happen. Limited funds. Coags normal. Thanks guys!
Go for ursodeoxycholic,
Go for ursodeoxycholic, gastric protectants (omeprazole, sulcralfate), anti-emetics, liver diet.
Thanks Remo!
Thanks Remo!