I totally agree: It’s a VSD (membranous) causing mild LV volume overload. Prognosis is always difficult in these guys, some of these live quite along time without any problems.
There’s 2 ways to go:
1) Monitor the dog (rescan in 3 dn 6 months) and once LV overload increases, consider pulmonary artery banding (creates a pulmonic stenosis to reduce shunt volume and avoid left sided failure as well as progression of PHT)
2) If interventional closure is an option, i would recommend the University of Gießen (Prof M. Schneider) who is likely most experienced with this
3) surgical closure is not optimal because of AV-Block risk (vicinity of the bundle of His).
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Hi!
I totally agree: It’s a
Hi!
I totally agree: It’s a VSD (membranous) causing mild LV volume overload. Prognosis is always difficult in these guys, some of these live quite along time without any problems.
There’s 2 ways to go:
1) Monitor the dog (rescan in 3 dn 6 months) and once LV overload increases, consider pulmonary artery banding (creates a pulmonic stenosis to reduce shunt volume and avoid left sided failure as well as progression of PHT)
2) If interventional closure is an option, i would recommend the University of Gießen (Prof M. Schneider) who is likely most experienced with this
3) surgical closure is not optimal because of AV-Block risk (vicinity of the bundle of His).
Regards,
Peter
Thank you Peter, really great
Thank you Peter, really great advice.